A new study reveals that astrocyte-derived GABA, not neuronal activity, plays a central role in PTSD by impairing the brain’s ability to extinguish traumatic memories.

GBA1 mutations impair ciliary Hedgehog signaling in Parkinson's disease, reducing neuroprotective factors and highlighting a ...

ACP-01 (VesCell™) produces signals like IL-8, VEGF, and angiogenin. They attract natural killer (NK) cells that suppress ...

Observational and philosophical analysis continued to examine the brain, as well as the impact that thinking, exercises, and ...

Researchers discovered that PTSD may be driven by excess GABA from astrocytes, not neurons. This chemical imbalance disrupts ...

The new drug has already passed Phase 1 safety trials in humans, making it a "strong candidate" for future PTSD treatments.

This study reports important negative results by showing that genetic removal of the RNA-binding protein PTBP1 in astrocytes is not sufficient to induce their conversion into neurons, challenging ...

A collaborative study between Dana-Farber Cancer Institute and the Broad Institute found that 8.9% of children with glioma, ...

This is a valuable study that explores the role of the conserved transcription factor POU4-2 in the maintenance, regeneration, and function of planarian mechanosensory neurons. The authors provide ...