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The epithelial–mesenchymal transition (EMT) is a cellular transdifferentiation program that enables epithelial cancer cells to acquire traits of high-grade malignancy, notably invasive and ...
The phenomenon of epithelial–mesenchymal transition (EMT) has gained attention in the field of cancer biology for its potential contribution to the progression of carcinomas. Tumor EMT is a ...
The following is a summary of “Cytokeratin 17 activates AKT signaling to induce epithelial-mesenchymal transition and promote bladder cancer progression,” published in the April 2025 issue of BMC ...
The developmental program Epithelial-Mesenchymal Transition (EMT) is frequently re-activated in metastatic and recurrent tumors. Our work provided a conceptual understanding depicting a causal role ...
One key feature of metastasis, Dr. Brekken explained, is epithelial-to-mesenchymal transition (EMT), in which cancer cells ...
4d
News-Medical.Net on MSNLipid metabolism as a driver of therapy resistance in triple negative breast cancerLipid metabolism has emerged as a central player in the progression and therapy resistance of breast cancer, particularly the aggressive subtype known as triple-negative breast cancer (TNBC).
Cancer cells can alter their identity through epithelial-mesenchymal transition (EMT), a process that enhances their ability ...
Background Muscle wasting and weakness (sarcopenia) are commonly associated with COPD causing frailty and reduced quality of ...
These processes are called the epithelial-to-mesenchymal transition (EMT) and the reverse mesenchymal-epithelial-transition (MET), and they enable cells to reversibly switch between stationary and ...
ENO2 overexpression promoted the migration and invasion of HNSCC cells both in vitro and in vivo, and this effect was mediated by the epithelial-mesenchymal (EMT) transition. Further studies ...
We currently have an inventory of over 290 validated antibodies that cover multiple total proteins and phosphoproteins in the following protein pathways or functional protein groups: ...
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